human tissue cdnas (TaKaRa)
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human tissue cdnas
Human Tissue Cdnas, supplied by TaKaRa, used in various techniques. Bioz Stars score: 95/100, based on 605 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+tissue+cdnas/Human+MTC+Panel+I/pmc11961601__41413_2025_415_MOESM1_ESM-44-35-41
Average 95 stars, based on 605 article reviews
Human Tissue Cdnas, supplied by TaKaRa, used in various techniques. Bioz Stars score: 95/100, based on 605 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+tissue+cdnas/Human+MTC+Panel+I/pmc11961601__41413_2025_415_MOESM1_ESM-44-35-41
Average 95 stars, based on 605 article reviews
human tissue cdnas - by Bioz Stars,
2026-10
95/100 stars
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Expressing:Article Title: KIAA1199 (CEMIP) regulates adipogenesis and whole-body energy metabolism Article Snippet: .. KIAA1199 highly expresses in bone and bone marrow stromal stem cells, and its expression and secretion are significantly reduced during adipocyte (AD) Fig.S1 differentiation in hBMSCs. (a) Relative expression levels of KIAA1199 mRNA in different Article Title: Discovery of NOvel CIP2A VAriant (NOCIVA) and its clinical relevance in myeloid leukemias Article Snippet: .. First, NOCIVA and CIP2A expression was analyzed in a panel of normal Article Title: CIP2A variant and uses thereof Article Snippet: .. Next we assessed expression of CIP2A and NOCIVA in a panel of normal Real-time Polymerase Chain Reaction:Article Title: KIAA1199 (CEMIP) regulates adipogenesis and whole-body energy metabolism Article Snippet: .. KIAA1199 highly expresses in bone and bone marrow stromal stem cells, and its expression and secretion are significantly reduced during adipocyte (AD) Fig.S1 differentiation in hBMSCs. (a) Relative expression levels of KIAA1199 mRNA in different Article Title: RETRACTED: Deficiency of a novel gene, Yulink, predisposes to heart failure and ventricular arrhythmia Article Snippet: Heart failure is amajor cardiovascular disease and is associatedwith significantmorbidity andmortality.. Sudden cardiacdeath (SCD) in heart failure is a disastrous cardiovascular phenomenon.However, few studies have examined the genetic background that determines susceptibility to heart failure andSCD.We found that deficiency of anewly identifiedgene,Yulink, promoted cardiacalternans in zebrafish cardiomyocytes, andgenetic knockdown (KD) resulted in pericardial edema, decreased cardiac output, and premature ventricular contractions.. Yulink KD morphants exhibited irregular action potentials, slower Ca reuptake, and alternans of Ca transients and action potential duration, which are hallmarks for SCD susceptibility in heart failure. Amplification:Article Title: Enzyme Activities of the Ceramide Synthases CERS2–6 Are Regulated by Phosphorylation in the C-terminal Region Article Snippet: .. Human CERS1 , - 5 , and - 6 cDNAs were amplified by PCR using Article Title: Enzyme Activities of the Ceramide Synthases CERS2–6 Are Regulated by Phosphorylation in the C-terminal Region Article Snippet: .. Human CERS1, 5, and 6 cDNAs were amplified by PCR using Polymerase Chain Reaction:Article Title: Enzyme Activities of the Ceramide Synthases CERS2–6 Are Regulated by Phosphorylation in the C-terminal Region Article Snippet: .. Human CERS1 , - 5 , and - 6 cDNAs were amplified by PCR using Article Title: Enzyme Activities of the Ceramide Synthases CERS2–6 Are Regulated by Phosphorylation in the C-terminal Region Article Snippet: .. Human CERS1, 5, and 6 cDNAs were amplified by PCR using SYBR Green Assay:Article Title: RETRACTED: Deficiency of a novel gene, Yulink, predisposes to heart failure and ventricular arrhythmia Article Snippet: Heart failure is amajor cardiovascular disease and is associatedwith significantmorbidity andmortality.. Sudden cardiacdeath (SCD) in heart failure is a disastrous cardiovascular phenomenon.However, few studies have examined the genetic background that determines susceptibility to heart failure andSCD.We found that deficiency of anewly identifiedgene,Yulink, promoted cardiacalternans in zebrafish cardiomyocytes, andgenetic knockdown (KD) resulted in pericardial edema, decreased cardiac output, and premature ventricular contractions.. Yulink KD morphants exhibited irregular action potentials, slower Ca reuptake, and alternans of Ca transients and action potential duration, which are hallmarks for SCD susceptibility in heart failure. |
